Peptide Protocol

NAD+: the cellular energy coenzyme — longevity from within.

Nicotinamide adenine dinucleotide. Injectable protocol for mitochondrial function, DNA repair, and cellular energy. IV, IM, or SubQ.

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250–500 mg
Typical Dose
4–12 weeks
Cycle
IV / IM / SubQ
Route
Daily or 2–3×/week
Frequency
Overview

What is NAD+?

NAD+ (nicotinamide adenine dinucleotide) is a dinucleotide coenzyme found in every cell of the human body. It plays a central role in redox reactions powering the electron transport chain and ATP synthesis, and serves as a required cofactor for sirtuins (SIRT1–7) — the longevity-associated deacylases linked to caloric restriction and lifespan extension in animal models.

NAD+ levels decline approximately 50% between age 20 and 50, a trajectory associated with mitochondrial dysfunction, increased DNA damage, and metabolic decline. Research has explored whether restoring NAD+ levels via direct injection (bypassing gut absorption) can recapitulate benefits observed in preclinical studies. While oral precursors (NMN, NR) are widely available, injectable NAD+ provides more direct tissue delivery with faster onset — with onset characteristics studied in early clinical investigations.

Mitochondrial Function
NAD+ is the rate-limiting substrate for Complex I of the electron transport chain. Restoring NAD+ directly supports mitochondrial ATP production and biogenesis.
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Sirtuin Activation
Sirtuins (SIRT1–7) require NAD+ as a cofactor. Elevated NAD+ amplifies sirtuin-mediated deacetylation, linked to metabolic regulation, stress resistance, and longevity.
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DNA Repair (PARP)
PARP enzymes consume NAD+ to repair DNA strand breaks. Adequate NAD+ availability is essential for efficient genomic maintenance under oxidative stress.
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Neuroprotection
NAD+ supports neuronal energy demands and has been studied for cognitive function, neuroinflammation reduction, and potential application in neurodegenerative research.
Dosing Protocol

Dosing Schedule

Parameters documented in published preclinical and clinical research.

⚠️ Research use only. The following documents parameters from published preclinical and clinical research. Not medical advice. Not for human consumption. Consult a licensed healthcare professional before any use.
ProtocolDoseFrequencyRouteNotes
Conservative start100–250 mgOnce dailyIV slow push / IMBegin low to assess flush response. IV onset is rapid; dilute in 100 mL saline and push over 1–2 hrs.
Standard250–500 mgDaily or 2–3×/weekIV / IM / SubQMost commonly reported research dose range. IM and SubQ offer comparable bioavailability with easier administration.
High dose500–1000 mgDaily (loading phase)IV (diluted)Used in some clinical NAD+ infusion protocols. Must be diluted and administered slowly to minimise side effects.
Maintenance100–250 mg2–3×/weekIM / SubQAfter initial loading, lower frequency maintenance is sufficient for most research purposes.
Safety Profile

Safety & Side Effects

✓ Generally Well Tolerated
Endogenous coenzyme — naturally present in all human cells
Extensive clinical trial data from NMN and NR supplementation research
No significant adverse effects at standard research doses
Both IV and SubQ/IM routes well-documented in clinical protocols
⚠ Potential Concerns
IV bolus without dilution causes flushing, nausea, and chest tightness — always dilute and infuse slowly
Theoretical concern around NAD+ and proliferating cells — not clearly established in clinical data
High-dose IV may cause transient hypotension
Long-term injectable NAD+ human safety data remains limited
⚠️
Research use onlyThis page is an educational reference. None of this constitutes medical advice. Consult a qualified professional before any use. All compounds are for research purposes only.
Evidence Base

Academic References

  1. [1]
    Yoshino J, et al. (2018). NAD+ Intermediates: The Biology and Therapeutic Potential of NMN and NR. Cell Metab. 27(3):513–528. PubMed ↗
  2. [2]
    Verdin E. (2015). NAD+ in aging, metabolism, and neurodegeneration. Science. 350(6265):1208–13. PubMed ↗
  3. [3]
    Rajman L, et al. (2018). Therapeutic Potential of NAD-Boosting Molecules: The In Vivo Evidence. Cell Metab. 27(3):529–547. PubMed ↗
  4. [4]
    Lautrup S, et al. (2019). NAD+ in Brain Aging and Neurodegenerative Disorders. Cell Metab. 30(4):630–655. PubMed ↗
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